1887

Abstract

induced a set of general stress proteins in response to a salt or heat stress. Cells subjected to a mild heat stress showed a protective response which enabled them to survive otherwise lethal temperatures (e.g. 52 °C). In a similar way bacteria were enabled to survive toxic concentrations of NaCl by pretreatment with lower salt concentrations. A mild heat shock induced a cross-protection against lethal salt stress. The pretreatment of cells with low salt, however, was less effective in the induction of thermotolerance than a preceding mild heat stress. Three stress proteins were identified on the basis of their N-terminal amino acid sequences as homologues of GroEL, DnaK and ClpP of . The role of general and specific stress proteins in the induction of thermotolerance/salt tolerance and cross-protection is discussed.

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/content/journal/micro/10.1099/00221287-138-10-2125
1992-10-01
2019-10-21
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http://instance.metastore.ingenta.com/content/journal/micro/10.1099/00221287-138-10-2125
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