1887

Abstract

A dominant mutant of impaired in aflatoxin production, designated and linked to in linkage group VII, was recovered after treatment of conidia with -methyl-′-nitro--nitrosoguanidine. Mutant failed to complement other non-allelic aflatoxin mutants in diploids and also restricted aflatoxin accumulation when paired with toxigenic strains in diploids. No detectable effect of was demonstrated in heterokaryons. This gene locus is apparently not involved in the extracellular degradation of aflatoxin.

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/content/journal/micro/10.1099/00221287-118-1-279
1980-05-01
2021-05-15
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