1887

Abstract

SUMMARY: DNA repair and genetic recombination have been studied in mutants of in which expression of inducible, SOS repair activities was altered by additional mutations either in or and appeared to act additively to increase sensitivity to UV irradiation and to reduce recombination proficiency. The defect was suppressed in strains carrying the allele of but not in strains carrying mutations that increased synthesis of protein or which directly inactivated repressor. These and other data are interpreted to suggest that the defect is related to a reduced activity of protein. The implications of these findings are discussed in relation to the control of SOS activities and cell division during normal growth.

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/content/journal/micro/10.1099/00221287-129-3-681
1983-03-01
2020-01-18
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http://instance.metastore.ingenta.com/content/journal/micro/10.1099/00221287-129-3-681
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