Two influenza A viruses, neurotropic and its recombinant are capable of forming plaques on BHK21 cells. plaques are larger than plaques. The small plaque size of virus is related to a slower rate of increase of than virus during many cycles of infection, not to inhibition by the non-cellular environment. The slow increase could be accounted for by a delay in the formation of infectious virus in each cycle accompanied by a simultaneous delay in release. The synthesis of virus is accompanied by a large quantity of unassembled or unstable non-infectious haemagglutinin; the synthesis of virus yields assembled infectious haemagglutinin predominantly.


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