1887

Abstract

Two independent mutants defective in glutathione reductase (EC 1.6.4.2) were isolated in an K12 strain lysogenized with bacteriophage Mu. The prophage was lost (and the ability to reduce glutathione regained) by 32% of the xylose-positive transductants when T4GT7 was used as the vector, but the markers were not cotransduced by P1. Similarly, the prophage site and were cotransduced by T4GT7 but not by P1. The gene maps between min 77 and 78 on the genome, and the mutation causes no growth defect.

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/content/journal/micro/10.1099/00221287-128-7-1631
1982-07-01
2024-04-26
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