1887

Abstract

Mutants of EBF 65/65 that could not grow on intermediates of the mandelate or benzyl alcohol pathways were isolated and in some cases the enzymic lesions were identified. Several catabolic markers were mapped using the plasmid pAVl. The mandelate genes appeared to be clustered near the auxotrophic marker but were not all contiguous with each other. The gene responsible for the appearance of the novel (+)-mandelate dehydrogenase appeared to be close to a gene responsible for the activity of the original (−)-mandelate dehydrogenase.

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1984-11-01
2024-04-25
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