Glucosamine acted as a gratuitous catabolite repressor of maltose utilization in This repression was relieved in a linear manner as the maltose concentration was increased. Three mutants were isolated in which maltose utilization was no longer repressed by glucosamine. One of these mutants may be generally deficient in catabolite repression since it was not defective in glucosamine transport but was insensitive to glucosamine repression on all catabolite repressible carbon sources tested. Each mutant possessed a maltose uptake system which was notably less sensitive to glucose repression than that of the wild-type strain. These results are discussed in terms of a proposed model for regulation of maltose utilization in


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