1887

Abstract

R plasmids pMG1, R2, R931 and pMG15 increased the survival of exposed to ultraviolet radiation (u.v.) in the wild type, and and mutants but did not alter the u.v.-response of a mutant. The R plasmid RPL11 reduced u.v.-survival in the wild type, and and mutants but did not alter the u.v.-response of a host. All the plasmids enhanced the level of spontaneous and u.v.-induced back mutation (Trp) in a strain.

The effect of a sublethal concentration of sodium arsenite following u.v.-irradiation was examined. It was concluded that in strains (pMG1) and (R931), u.v.-protection is determined by a -dependent, arsenite-sensitive repair pathway, whereas in strains (R2) and (pMG15), u.v.-protection is determined by a -dependent, arsenite-insensitive step in DNA repair.

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/content/journal/micro/10.1099/00221287-108-1-119
1978-09-01
2019-10-16
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