strains differ in their sensitivity to some of the bactericidal lipids produced by the host in staphylococcal abscesses. To evaluate whether the presence of a capsule might account for these differences, capsulate and non-capsulate strains were compared for their sensitivity to staphylococcal abscess homogenates and the neutral lipid fraction derived from such material. Although the presence of a capsule appeared to reduce sensitivity, two non-capsulate mutants were only about three-to-four times more sensitive than their capsulate parent strains. Another strain, known for its resistance to these bactericidal lipids, was not capsulate. This suggests that mechanisms other than capsule formation must also determine sensitivity to the lipids.


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