1887

Abstract

Summary

The in-vitro susceptibility to host immune defence mechanisms of Campylobacter pyloridis was investigated. was sensitive to antibodydependent complement-mediated bactericidal activity of serum. The bacteria were phagocytosed and efficiently killed by polymorphonuclear neutrophils in the presence of serum opsonins. Serum opsonin depletion studies indicated that an intact classical complement pathway was required for optimal phagocytic killing.

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/content/journal/jmm/10.1099/00222615-23-3-233
1987-05-01
2019-10-21
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http://instance.metastore.ingenta.com/content/journal/jmm/10.1099/00222615-23-3-233
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