Status of the ICP34.5 gene in herpes simplex virus type 1 strain 17

References

1. Ackermann M., Chou J., Sarmiento M., Lerner R. A., Roizman B. 1986; Identification by antibody to a synthetic peptide of a protein specified by a diploid gene located in the terminal repeats of the L component of herpes simplex virus genome. Journal of Virology 58:843–850
   [Google Scholar]
2. Chou J., Roizman B. 1986; The terminal a sequence of the herpes simplex virus genome contains the promoter of a gene located in the repeat sequences of the L component. Journal of Virology 57:629–637
   [Google Scholar]
3. Chou J., Roizman B. 1990; The herpes simplex virus 1 gene for ICP34.5, which maps in inverted repeats, is conserved in several limited-passage isolates but not in strain 17syn⁺ . Journal of Virology 64:1014–1020
   [Google Scholar]
4. Chou J., Kern E. R., Whitley R. J., Roizman B. 1990; Mapping of herpes simplex virus-1 neurovirulence to γ ₁ 34.5, a gene nonessential for growth in culture. Science 250:1262–1265
   [Google Scholar]
5. McGeoch D. J., Barnett B. C. 1991; Neurovirulence factor. Nature, London 353:609
   [Google Scholar]
6. McGeoch D. J., Dolan A., Donald S., Rixon F. J. 1985; Sequence determination and genetic content of the short unique region in the genome of herpes simplex virus type 1. Journal of Molecular Biology 181:1–13
   [Google Scholar]
7. McGeoch D. J., Dalrymple M. A., Davison A. J., Dolan A., Frame M. C., McNab D., Perry L. J., Scott J. E., Taylor P. 1988; The complete DNA sequence of the long unique region in the genome of herpes simplex virus type 1. Journal of General Virology 69:1531–1574
   [Google Scholar]
8. McGeoch D. J., Cunningham C., McIntyre G., Dolan A. 1991; Comparative sequence analysis of the long repeat regions and adjoining parts of the long unique regions in the genomes of herpes simplex viruses types 1 and 2. Journal of General Virology 72:3057–3075
   [Google Scholar]
9. MacLean A. R., Fareed M. U., Robertson L., Harland J., Brown S. M. 1991; Herpes simplex virus type 1 deletion variants 1714 and 1716 pinpoint neurovirulence-related sequences in Glasgow strain 17⁺ between immediate early gene 1 and the ‘a’ sequence. Journal of General Virology 72:631–639
   [Google Scholar]
10. Perry L. J., McGeoch D. J. 1988; The DNA sequences of the long repeat region and adjoining parts of the long unique region in the genome of herpes simplex virus type 1. Journal of General Virology 69:2831–2846
    [Google Scholar]
11. Taha M. Y., Clements G. B., Brown S. M. 1989a; A variant of herpes simplex virus type 2 strain HG52 with a 1.5 kb deletion in R_L between 0 to 0.02 and 0.81 to 0.83 map units is non-neurovirulent for mice. Journal of General Virology 70:705–716
    [Google Scholar]
12. Taha M. Y., Brown S. M., Clements G. B. 1989b; The herpes simplex virus type 2 (HG52) variant JH2604 has a 1488 bp deletion which eliminates neurovirulence in mice. Journal of General Virology 70:3073–3078
    [Google Scholar]
13. Thompson R. L., Wagner E. K., Stevens J. G. 1983; Physical location of a herpes simplex virus type-1 function(s) specifically involved with a 10 million-fold increase in HSV neurovirulence. Virology 131:180–192
    [Google Scholar]
14. Thompson R. L., Rogers S. K., Zerhusen M. A. 1989; Herpes simplex virus neurovirulence and productive infection of neural cells is associated with a function which maps between 0.82 and 0.832 map units on the HSV genome. Virology 172:435–450
    [Google Scholar]