Virological Aspects of Measles Virus-induced Encephalomyelitis in Lewis and BN Rats

References

1. Baczko K., Liebert U. G., Billeter M., Cattaneo R., Budka H., Ter Meulen V. 1986; Expression of defective measles virus genes in brain tissues of patients with subacute sclerosing panencephalitis. Journal of Virology 59:472–478
   [Google Scholar]
2. Carter M. J., Willcocks M. M., Löffler S., Ter Meulen V. 1982; Relationships between monoclonal antibody-binding sites on the measles virus haemagglutinin. Journal of General Virology 63:113–120
   [Google Scholar]
3. Carter M. J., Willcocks M. M., Löffler S., Ter Meulen V. 1983; Comparison of lytic and persistent measles virus matrix proteins by competition radioimmunoassay. Journal of General Virology 64:1801–1805
   [Google Scholar]
4. Cattaneo R., Schmid A., Rebmann G., Baczko K., Ter Meulen V., Bellini W. J., Rozenblatt S., Billeter M. A. 1986; Accumulated measles mutations in a case of subacute sclerosing panencephalitis: interrupted matrix protein reading frame and transcription alterations. Virology 154:97–107
   [Google Scholar]
5. Gasser D. L., Newlin C. M., Palm J., Gonatas N. K. 1973; Genetic control of susceptibility to experimental allergic encephalomyelitis in rats. Science 181:872–873
   [Google Scholar]
6. Gendelman H. E., Wolinsky J. S., Johnson R. T., Pressman N. J., Pezeshkpour G. H., Boisset G. F. 1984; Measles encephalomyelitis: lack of evidence of viral invasion of the central nervous system and quantitative study of the nature of demyelination. Annals of Neurology 15:353–360
   [Google Scholar]
7. Johnson K. P., Norrby E., Swoveland P., Carrigan D. R. 1981; Experimental subacute sclerosing panencephalitis: selective disappearance of measles virus matrix protein from the central nervous system. Journal of Infectious Diseases 144:161–169
   [Google Scholar]
8. Johnson R. T., Griffin D. E., Hjrsch R. L., Wolinsky J. S., Roedenbeck S., De Soriano I. L. 1984; Measles encephalomyelitis: clinical and immunological studies. New England Journal of Medicine 310:137–141
   [Google Scholar]
9. Kaerber G. 1931; Beitrag zur kollektiven Behandlung pharmakologischer Reihenversuche. Archiv für experimented Pathologie und Pharmakologie 162:480–492
   [Google Scholar]
10. Kornblum J. 1968; The application of the irradiated hamster test to the study of experimental allergic encephalomyelitis. Journal of Immunology 101:702–710
    [Google Scholar]
11. Kristensson K., ÖRvell C., Leestma J., Norrby E. 1983; Sendai virus infection in the brains of mice: distribution of viral antigens studied with monoclonal antibodies. Journal of Infectious Diseases 147:297–301
    [Google Scholar]
12. Kristensson K., Örvell C., Gmalm G., Norrby E. 1984; Mumps virus infection of a developing mouse brain. Appearance of structural virus proteins demonstrated with monoclonal antibodies. Journal of Neuropathology and Experimental Neurology 43:131–140
    [Google Scholar]
13. Liebert U. G., Baczko K., Budka H., Ter Meulen V. 1986; Restricted expression of measles virus proteins in brains from cases of subacute sclerosing panencephalitis. Journal of General Virology 67:2435–2444
    [Google Scholar]
14. Miller C. A., Carrigan D. R. 1982; Reversible repression and activation of measles virus infection in neural cells. Proceedings of the National Academy of Sciences U.S.A.: 791629–1633
    [Google Scholar]
15. Miller H. G., Stanton J. B., Gibbons J. L. 1956; Parainfectious encephalomyelitis and related syndromes. Quarterly Journal of Medicine 25:427–505
    [Google Scholar]
16. Morgan E. M., Rapp F. 1977; Measles virus and its associated diseases. Bacteriological Reviews 41:636–666
    [Google Scholar]
17. Norrby E., Chen S.-N., Togashi T., Sheshberadaran H., Johnson K. P. 1982; Five measles virus antigens demonstrated by use of mouse hybridoma antibodies in productively infected tissue culture cells. Archives of Virology 71:1–11
    [Google Scholar]
18. Percy D, Coulter-Mackie M. 1982; Measles virus encephalitis and retinopathy in the Wistar rat. Experimental and Molecular Pathology 36:435–446
    [Google Scholar]
19. Rammohan K. W., Dubois-Dalcq M., Rentier B., Paul J. 1983; Experimental models to study measles virus persistence in the nervous system. In Progress in Neuropathology 5 pp 113–137 Zimmermann H. M. Edited by New York: Raven Press;
    [Google Scholar]
20. Sato T. A., Fukuda A., Sugiura A. 1985; Characterization of major structural proteins of measles virus with monoclonal antibodies. Journal of General Virology 66:1397–1409
    [Google Scholar]
21. Schumacher H. P., Albrecht P., Tauraso N. M. 1972; The effect of altered immune reactivity on experimental measles encephalitis in rats. Archiv für die gesamte Virusforschung 37:218–229
    [Google Scholar]
22. Sheppard R. D., Raine C. S., Bornstein M. B., Udem S. A. 1985; Measles virus matrix protein synthesized in a subacute sclerosing panencephalitis cell line. Science 228:1219–1221
    [Google Scholar]
23. Sheshberadaran H., Chen S.-N., Norrby E. 1983; Monoclonal antibodies against five structural components of measles virus. I. Characteristics of antigenic determinants of nine strains of measles virus. Virology 128:341–353
    [Google Scholar]
24. Ter Meulen V., Carter M. J. 1982; Morbillivirus persistent infections in animals and man. In Virus Persistence pp 97–132 Mahy B. W. J., Minson A. C., Darby G. K. Edited by Cambridge: Cambridge University Press;
    [Google Scholar]
25. Ter Meulen V., Stephenson J. R., Kreth H. W. 1983; Subacute sclerosing panencephalitis. In Comprehensive Virology 18 pp 105–159 Fraenkel-Conrat H., Wagner R. R. Edited by New York: Plenum Press;
    [Google Scholar]
26. Watanabe R., Wege H., Ter Meulen V. 1983; Adoptive transfer of EAE-like lesions from rats with coronavirus-induced demyelinating encephalomyelitis. Nature; London: 305150–153
    [Google Scholar]
27. Yoshikawa Y., Yamanouchi K. 1984; Effect of papaverine treatment on replication of measles virus in human neural and non-neural cells. Journal of Virology 50:489–496
    [Google Scholar]
28. Young K. K. Y., Heinke B. E., Wechsler S. L. 1985; M protein instability and lack of M protein processing associated with non-productive persistent infection of HeLa cells by measles virus. Virology 143:536–545
    [Google Scholar]