Biochemical Analysis of Rabies Virus Proteins in Three Persistently Infected BHK-21 Cell Lines

References

1. Blondel D., Dezelee S., Wyers F. 1983; Vesicular stomatitis virus growth in Drosophila melanogaster cells. II. Modifications of viral protein phosphorylation. Journal of General Virology 64:1793–1799
   [Google Scholar]
2. Bradford M. M. 1976; A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding. Analytical Biochemistry 72:248–254
   [Google Scholar]
3. Capco D. G., Wan K., Penman S. 1982; The nuclear matrix: three dimensional architecture and protein composition. Cell 29:847–858
   [Google Scholar]
4. Dietzschold B., Cox J. H., Schneider L. G. 1979; Rabies virus strains: a comparison study by polypeptide analysis of vaccine strains with different pathogenic patterns. Virology 98:63–75
   [Google Scholar]
5. Ehrnst A. 1979; Growth phase related loss of measles virus surface-associated antigens and cytotoxic susceptibility in persistently infected cells. Journal of General Virology 45:547–556
   [Google Scholar]
6. Flamand A., Wiktor T. J., Koprowski H. 1980a; Use of hybridoma monoclonal antibodies in the detection of antigenic differences between rabies and rabies-related virus proteins. I. The nucleocapsid protein. Journal of General Virology 48:97–104
   [Google Scholar]
7. Flamand A., Wiktor T. J., Koprowski H. 1980b; Use of hybridoma monoclonal antibodies in the detection of antigenic differences between rabies and rabies-related virus proteins. II. The glycoprotein. Journal of General Virology 48:105–109
   [Google Scholar]
8. Grabau E. A., Holland J. J. 1982; Analysis of viral and defective-interfering nucleocapsids in acute and persistent infection by rhabdoviruses. Journal of General Virology 60:87–97
   [Google Scholar]
9. Hall W. W., Choppin P. W. 1979; Evidence for lack of synthesis of the M polypeptide of measles virus in brain cells in subacute panencephalitis. Virology 99:443–447
   [Google Scholar]
10. Holland J. J., Villarreal L. P., Welsh R. M., Oldstone M. B. A., Kohne D., Lazzarini R., Scolnick E. 1976; Long-term persistent vesicular stomatitis virus and rabies virus infection of cells in vitro. Journal of General Virology 33:193–211
    [Google Scholar]
11. Holland J. J., Grabau E. A., Jones C. L., Semler B. L. 1979; Evolution of multiple genome mutations during long-term persistent infection by vesicular stomatitis virus. Cell 16:495–504
    [Google Scholar]
12. Holland J. J., Kennedy S. I. T., Semler B. L., Jones C. L., Roux L., Grabau E. A. 1980; Defective interfering RNA viruses and the host-cell response. In Comprehensive Virology vol 16: pp 137–192 Edited by Fraenkel-Conrat H., Wagner R. R. New York: Plenum Press;
    [Google Scholar]
13. Kawai A., Matsumoto S., Tanabe K. 1975; Characterization of rabies virus recovered from persistently infected BHK cells. Virology 67:520–533
    [Google Scholar]
14. Laemmli U. K. 1970; Cleavage of structural proteins during the assembly of the head of bacteriophage T4. Nature, London 221:680–685
    [Google Scholar]
15. Lafay F., Benejean J. 1981; Temperature sensitive mutants of vesicular stomatitis virus: tryptic peptide maps of the proteins modified in complementation groups II and IV. Virology 111:93–102
    [Google Scholar]
16. Lafon M., Wiktor T. J., Macfarlan R. I. 1983; Antigenic sites on the CVS rabies virus glycoprotein: analysis with monoclonal antibodies. Journal of General Virology 64:843–851
    [Google Scholar]
17. Lehmann-Grube F., Martinez-Peralta L., Bruns M., Lohler J. 1983; Persistent infection of mice with lymphocytic choriomeningitis virus. In Comprehensive Virology vol 18: pp 43–103 Edited by Fraenkel-Conrat H., Wagner R. R. New York: Plenum Press;
    [Google Scholar]
18. Lin F. H., Thormar H. 1980; Absence of M protein in a cell-associated subacute sclerosing panencephalitis virus. Nature, London 285:490–492
    [Google Scholar]
19. Machamer C. E., Hayes E. C., Zweerink H. J. 1981; Cells infected with a cell-associated SSPE virus do not express M protein. Virology 108:515–520
    [Google Scholar]
20. Macpherson L., Stoker M. 1962; Polyoma transformation of hamster clones. An investigation of genetic factors affecting cell competence. Virology 16:147–151
    [Google Scholar]
21. Martinet-Edelist C., Deutsch V., Tuffereau C., Genty N. 1984; Intracellular events following the infection of different cell types with vesicular stomatis virus subviral particles. Virology 135:266–278
    [Google Scholar]
22. Oldstone M. B. A., Buchmeier M. J. 1982; Restricted expression of viral glycoprotein in cells of persistently infected mice. Nature, London 300:360–362
    [Google Scholar]
23. Roux L., Waldvogel F. A. 1982; Instability of the viral M protein in BHK-21 cells persistently infected with Sendai virus. Cell 28:293–302
    [Google Scholar]
24. Rowlands D., Grabau E. A., Spindler K., Jones C., Semler B., Holland J. J. 1980; Virus protein changes and RNA termini alterations evolving during persistent infection. Cell 19:871–880
    [Google Scholar]
25. Semler B. L., Holland J. J. 1979; Persistent vesicular stomatitis virus infection mediates base substitutions in viral RNA termini. Journal of Virology 32:420–428
    [Google Scholar]
26. Ter Meulen V., Carter M. J. 1982; Morbillivirus persistent infection in animals and man. In Virus Persistence pp 97–132 Edited by Mahy B. W. J., Minson A. C., Darby G. K. Cambridge: Cambridge University Press;
    [Google Scholar]
27. Ter Meulen V., Stephenson J. R., Kreth H. W. 1983; Subacute sclerosing panencephalitis. In Comprehensive virology vol 18: pp 105–159 Edited by Fraenkel-Conrat H., Wagner R. R. New York: Plenum Press;
    [Google Scholar]
28. Tuffereau C., Flamand A. 1983; Characterization of rabies virus production by five persistently infected BHK-21 cell lines. Annales de Virologie 134E:505–522
    [Google Scholar]
29. Villarreal L. P., Holland J. J. 1976; RNA synthesis in BHK 21 cells persistently infected with vesicular stomatitis virus and rabies virus. Journal of General Virology 33:213–224
    [Google Scholar]
30. Wechsler S. L., Fields B. N. 1978; Differences between the intracellular polypeptides of measles and subacute sclerosing panencephalitis virus. Nature, London 272:458–460
    [Google Scholar]
31. Wild T. F., Bijlenga G. 1981; A rabies virus persistent infection inBHK21 cells. Journal of General Virology 51:169–177
    [Google Scholar]
32. Youngner I. S., Preble O. T. 1980; Viral persistence: evolution of viral populations. In Comprehensive Virology vol 16: pp 73–125 Edited by Fraenkel-Conrat H., Wagner R. R. New York: Plenum Press;
    [Google Scholar]