Analysis of Viral and Defective-Interfering Nucleocapsids in Acute and Persistent Infection by Rhabdoviruses

References

1. Clark H. F., Ohtani S. 1976; Temperature-sensitive mutants of rabies virus in mice: a mutant (ts2) revertant mixture selectively pathogenic by the peripheral route of inoculation. Infection and Immunity 13:1418–1425
   [Google Scholar]
2. Clark H. F., Parks N. F., Wunner W. H. 1981; Defective interfering particles of fixed rabies viruses: lack of correlation with attenuation of auto-interference in mice. Journal of General Virology 52:245–258
   [Google Scholar]
3. Crick J., Brown F. 1974; An interfering component of rabies virus which contains RNA. Journal of General Virology 22:147–151
   [Google Scholar]
4. Fields B. N., Raine C. S. 1974; Altered neurologic disease induced by reovirus mutants. In Mechanisms of Virus Disease vol 1: pp 161–167 Edited by Fox W. X., Fox C. F. Menlo Park, California: W. A. Benjamin;
   [Google Scholar]
5. Holland J. J., Villarreal L. P. 1974; Persistent noncytocidal vesicular stomatitis virus infections mediated by defective T particles that suppress virion transcriptase. Proceedings of the National Academy of Sciences of the United States of America 71:2956–2960
   [Google Scholar]
6. Holland J. J., Villarreal L. P. 1975; Purification of defective interfering T particles of vesicular stomatitis and rabies virus generated in vitro in brains of newborn mice. Virology 67:438–449
   [Google Scholar]
7. Holland J. J., Villarrreal L. P., Breindl M. 1976a; Factors involved in the generation and replication of rhabdovirus defective interfering T particles. Journal of Virology 17:805–815
   [Google Scholar]
8. Holland J. J., Villarreal L. P., Welsh R. M., Oldstone M. B. A., Kohne D., Lazzarini R., Scolnick E. 1976b; Long-term persistent vesicular stomatitis virus and rabies virus infection of cells in vitro. Journal oj General Virology 33:193–211
   [Google Scholar]
9. Holland J. J., Grabau E. A., Jones C. L., Semler B. L. 1979; Evolution of multiple genome mutations during long-term persistent infection by vesicular stomatitis virus. Cell 16:495–504
   [Google Scholar]
10. Holland J., Spindler K., Grabau E., Semler B., Jones C., Horodyski F., Rowlands D., Janis B., Reid L., Minato N., Bloom B. 1980; Viral mutation in persistent infection. In Animal Virus Genetics pp 695–709 Edited by Fields B. N., Jaenisch J., Fox C. F. New York: Academic Press;
    [Google Scholar]
11. Horodyski F. M., Holland J. J. 1980; Viruses isolated from cells persistently infected with vesicular stomatitis virus show altered interactions with defective interfering particles. Journal of Virology 36:627–631
    [Google Scholar]
12. Huang A. S., Baltimore D. 1970; Defective viral particles and viral disease processes. Nature, London 226:325–327
    [Google Scholar]
13. Huang A. S., Manders E. K. 1972; Ribonucleic acid synthesis of vesicular stomatitis virus. IV. Transcription by standard virus in the presence of defective interfering particles. Journal of Virology 9:909–916
    [Google Scholar]
14. Kawai A. 1977; Transcriptase activity associated with rabies virion. Journal of Virology 24:826–835
    [Google Scholar]
15. Kawai A., Matsumoto S. 1977; Interfering and noninterfering particles generated by a rabies small plaque variant virus. Virology 76:60–71
    [Google Scholar]
16. Kawai A., Matsumoto S., Tanabe K. 1975; Characterization of rabies viruses recovered from persistently infected BHK cells. Virology 67:520–533
    [Google Scholar]
17. Khan S. R., Lazzarini R. A. 1977; The relationship between autointerference and the replication of a defective interfering particle. Virology 77:189–201
    [Google Scholar]
18. Laemmli U. K. 1970; Cleavage of structural proteins during the assembly of the head of bacteriophage T4. Nature, London 227:680–685
    [Google Scholar]
19. Lodmell D. L., Bell J. F., Moore J., Raymond G. H. 1969; Comparative study of abortive and nonabortive rabies in mice. Journal of Infectious Diseases 119:569–580
    [Google Scholar]
20. Mudd J. A., Summers D. F. 1970; Polysomal ribonucleic acid of vesicular stomatitis virus infected FleLa cells. Virology 42:958–968
    [Google Scholar]
21. Murphy F. A. 1977; Rabies pathogenesis (a brief review). Archives of Virology 54:279–297
    [Google Scholar]
22. Naito S., Matsumoto S. 1978; Identification of cellular actin within the rabies virus. Virology 91:151–163
    [Google Scholar]
23. Nishiyama Y. 1977; Studies of L cells persistently infected with VSV: factors involved in the regulation of persistent infection. Journal of General Virology 35:265–279
    [Google Scholar]
24. Nishiyama Y., Ito J., Shimokata D. 1978; Properties of viruses selected during persistent infection of L cells with VSV. Journal of General Virology 40:481–484
    [Google Scholar]
25. Palma E. L., Huang A. 1974; Cyclic production of vesicular stomatitis virus caused by defective interfering particles. Journal of Infectious Diseases 126:402–410
    [Google Scholar]
26. Perrault J., Holland J. J. 1972; Absence of transcriptase activity and transcription-inhibiting ability in defective interfering particles of vesicular stomatitis virus. Virology 50:159–170
    [Google Scholar]
27. Preble O. T., Youngner J. S. 1973; Temperature-sensitive defect of mutants isolated from L cells persistently infected with Newcastle disease virus. Journal of Virology 12:472–480
    [Google Scholar]
28. Pringle C. R. 1970; Genetic characteristics of conditional lethal mutants of vesicular stomatitis virus induced by 5-fluorouracil, 5-azacytidine and ethylmethane sulfonate. Journal of Virology 5:559–567
    [Google Scholar]
29. Ramseur J. F., Friedman R. M. 1977; Prolonged infection of interferon-treated cells by vesicular stomatitis virus: possible role of temperature-sensitive mutants and interferon. Journal of General Virology 37:523–533
    [Google Scholar]
30. Roux L., Holland J. J. 1979; Role of defective interfering particles of Sendai virus in persistent infections. Virology 93:91–103
    [Google Scholar]
31. Roux L., Waldvogel F. A. 1981; Establishment of Sendai virus persistent infection: biochemical analysis of the early phase of a standard plus defective interfering virus infection of BF1K cells. Virology 112:400–410
    [Google Scholar]
32. Rowlands D., Grabau E., Spindler K., Jones C., Semler B., Holland J. 1980; Virus protein changes and RNA termini alterations evolving during persistent infection. Cell 19:871–880
    [Google Scholar]
33. Sokol F., Kuwert E., Wiktor T. J., Hummeler K., Koprowski H. 1968; Purification of rabies virus grown in tissue culture. Journal of Virology 2:836–849
    [Google Scholar]
34. Stevens J., Todaro B., Fox C. F. 1978 Persistent Viruses New York: Academic Press;
    [Google Scholar]
35. Villarreal L. P., Holland J. J. 1976; RNA synthesis in BHK_2I cells persistently infected with vesicular stomatitis and rabies virus. Journal of General Virology 33:213–224
    [Google Scholar]
36. Wiktor T. J., Clark H. F. 1972; Chronic rabies virus infection of cell cultures. Infection and Immunity 6:988–995
    [Google Scholar]
37. Wiktor T. J., Koprowski H., Rorke L. B. 1972; Localized rabies infection in mice. Proceedings of the Society for Experimental Biology and Medicine 140:759–764
    [Google Scholar]
38. Wiktor T. J., Deitzschold B., Leamnson R. N., Koprowski H. 1977; Induction and biological properties of defective interfering particles of rabies virus. Journal of Virology 21:626–635
    [Google Scholar]
39. Wunner W. H., Clark H. F. 1980; Regeneration of DI particles of virulent and attenuated rabies virus: genome characterization and lack of correlation with virulence phenotype. Journal of General Virology 51:69–81
    [Google Scholar]
40. Youngner J. S., Dubovi J., Quagliana D. O., Kelley M., Preble O. T. 1976; Role of temperature-sensitive mutants in persistent infections initiated with vesicular stomatitis virus. Journal of Virology 19:90–101
    [Google Scholar]