1887

Abstract

SUMMARY

The uptake of H-inositol into the pool of free inositol and its incorporation into the lipid phosphatidylinositol have been studied in various avian and mammalian cells infected by different viruses. In all the virus-cell systems investigated, virus infection results in a drastically reduced amount of free H-inositol about 3 to 5 h post-infection, demonstrable in the infected cells as compared to the mock-infected controls. In contrast, the incorporation of H-inositol into lipid can be enhanced, reduced, or not influenced at all, depending on the virus-cell system under observation.

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1978-08-01
2024-04-19
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