1887

Abstract

Summary

The multiplication of fowl-plague virus was much less sensitive to actinomycin D in chick cells infected with avian myeloblastosis virus and in hamster cells, transformed by Rous sarcoma virus than in comparable normal cultures. In addition, reproduction of fowl-plague virus was less effectively suppressed by u.v.-irradiation of cells carrying avian myeloblastosis or Rous sarcoma virus than of control cultures. These findings could indicate that avian myeloblastosis and Rous sarcoma viruses induce a function of host-cell DNA, whose products can be utilized in reproduction of fowl-plague virus.

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/content/journal/jgv/10.1099/0022-1317-4-4-571
1969-06-01
2019-10-18
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http://instance.metastore.ingenta.com/content/journal/jgv/10.1099/0022-1317-4-4-571
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